Potential Health Impacts of Gamma-Frequency Sound in Server Room
AHMED N. H. ALNUAIMY, RASHA HASHEM, MICHAEL JOHNSON
Department of Computer Techniques Engineering
Al-Rafidain University College
Baghdad | Palestine Street
IRAQ
Abstract: - Hearing degradation caused by an exposure to excessive noise is irreversible. Many of the other
relative hazards that can be developed from noise-induced hearing loss are impaired communication with
family and coworkers, social isolation, irritability, decreasing of self-esteem, anxiety, and loss of productivity.
Hearing impairment is a significant health hazard that is naturally occurs with aging. Tinnitus is a disorder in
hearing ability and can cause a ringing in the ear without a source for physical sound. More than 40 million
people in the United States are suffering from tinnitus disorder. Fourteen percent of adults are suffering from
chronic tinnitus, and 50% of normal adults with no clinically confirmed disorders in hearing ability experience
subtle tinnitus in a silent environment. An exposure to excessive noise and the process of natural aging of
people may increase the occurrence of Tinnitus. Tinnitus is a spontaneous auditory perception that is associated
with the continued activity of the gamma frequency band (30 Hz - 80Hz). Server Room can be considered as a
continuous source of gamma frequency. Server room running devices are generating a continuous noise that
most of its power is allocated in the band of gamma frequency.
Key-Words: - Noise, Server Room, OSHA, Gamma Frequency, Tinnitus.
Received: April 12, 2021. Revised: January 26, 2022. Accepted: February 24, 2022. Published: April 2, 2022.
1 Introduction
Large server rooms are considered as a new
workplace phenomenon and verify merit as an
emerging technology. Excessive noise exposure to
employees working with servers may be present in
data storage sites [1]. Corporate server rooms
provide a single location for residential computers
to support business goals, which are usually a small
temperature-controlled, secure room with a
minimum number of passengers. Maintaining a
small area allows greater access control (for
example, a high degree of security), and also puts
the servers in a more intense format.
The server is designed to deliver data to other
computers (clients) and process requests over the
Internet, or a local network [2]. Those rooms
contain multiple devices responsible for transmitting
data to and from these data center. These rooms
have different densities and dimensions depending
on the room’s architecture, brand of the server, the
server’s age, and racks height in addition to the
proximity of the units to each other. Strategic
placement of units can amplify sound in every
server room, as the sound pressure depends on the
number of servers and how well they are positioned
from each other. Proximity and density of servers
have the ability to produce excessive harmful noise.
Some operators may find the volume and frequency
uncomfortable, and excessive noise may be
dangerous to their well-being.
Sound parameters are pressure and frequency
that are measured in Newton/Square Meter (N/m2)
or Pascal (Pa) and (Hertz) respectively. Pressure and
frequency have health hazard on the users, but the
pressure of the sound is the only factor that is
regulated by OSHA (Occupational Safety and
Health Administration) [3]. Sound in server rooms
has a frequency resonant that may be offensive to
some people and can somehow cause health issue,
but unfortunately is not regulated by any of
governmental agency. OSHA mandated that sound
pressure levels should not exceed a certain level so
that they do not cause an occupational health issue
to employees. In this study, we evaluated and
investigated sound frequency as it may adversely
impact the users. For example, gamma frequency
(40 Hertz–100 Hertz) of the sound band has been
discovered to be uncomfortable or distracting for
some individuals [4]. The average hearing range of
the person is maintained from 20 to 20,000 Hertz.
The range of the frequency investigated in this study
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can be reviewed in the Result section of this paper.
The objective of this research was to investigate if
the sound generated by the servers caused
discomfort for the users that maintain and use those
data center and also to analyze the characteristics of
noise.
The purpose of this study was to find out how
much did the servers generate of sound (power and
frequency) in a specific area, and then evaluate
whether the sound levels at gamma frequency need
to be reduced to avoid health hazard or issues
related to the productivity for the users. The
preferred way to address occupational exposure is
through engineering methods, preferably in the
design process. If the room or equipment can be
configured in a manner that eliminates or reduces
occupational exposures to the workers in the
workplace, then this could be the first approach by
ergonomic engineer supposing that it is
economically feasible. Engineering solutions (i.e.,
workplace design to reduce or avoid workers
exposure to risk condition) do not necessitate
administrative controls such as reduction of the
work hours of employees exposed to a certain
contaminant (in this case, sound), rotation of
employees in a specific job position, or mandate
annual medical test for hearing and compliance by
wearing equipment for safety purposes. The
preferred approach for reduction of noise impact
should always involve engineering solution first,
then administrative controls and finally resort
protective personal equipment.
The personal usage of protective equipment may
require the documentation for hearing capabilities of
each employee, and an investigation of the sound
reduction required amount that is defined by OSHA
29CFR1910.95 Hearing Conservation to ensure
compliance with regulation [3]. Annually, the cost
of conducting tests in order to determine employees’
hearing capabilities to choose convenient personal
equipment for hearing protectively can be expensive
in the long run.
Acoustics can be achieved or reduced in several
different ways depending on room configurations,
equipment placed in rooms, floor/wall construction
restrictions, and sound reduction materials. As an
engineering solution, noise encapsulation potentially
reduces the sound by isolating the device that emit
the sound from the user by enclosing it, completely.
Sound reduction methods may still allow the
equipment to be accessed by the user easily and
keep the sound below harmful levels. Sound
reduction involves the usage of appropriate personal
protective equipment (ear muffs, ear plugs, etc.) if
the pressure levels of the sound exceed the values
defined by OSHA. From engineering perspective,
noise cancellation is considered as another method
of sound reduction and it is more effective in work
environment. Noise cancellation method is
consisting of devices that use a technology of signal
processing to reduce the noise by capturing the
noise signal and then emitting an inverting version
of the sound wave, it is thereby canceling some of
the noise waves. Other forms of sound reduction are
accomplished by utilizing acoustic materials on
floor, ceilings, or walls, and that reduce the pressure
of the noise that emitted by the devices [5]. Personal
Protective Equipment (PPE) is typically employed
by organizations when engineering and
administrative solutions do not minimize sound to
an acceptable level to comply with federal
regulations [1]. Administrative solutions to protect
the hearing of employees include rotating
employees reducing employee exposure to noise.
These solutions create challenges in the scheduling
for organizations and are therefore not as desirable
as reducing or eliminating the sound exposure.
Lastly, the use of earmuffs, earplugs or any other
similar PPE may adversely impact the workers
ability to communicate while they are in a server
room and cause errors during the work due to poor
communication [5].
2 Tinnitus
Tinnitus is hearing impairment that is typically
associated with damage that occurs due to noise
trauma or chronically noise exposure [4]. Such
damage can hurt the central auditory system,
specifically the neural synchrony within the central
auditory system. These changes have been reported
in various studies conducted for animals and
humans and could be the cause of various
pathologies [4]. These damages have been reported
to be specifically tied to the gamma range (30 Hz
80 Hz) of sound frequency [4]. Tinnitus has been
characterized as one of the common auditory
disorders in the population. The anatomical
substrates and disease associations still continue to
be defined. However, Semantic Dementia (SemD)
patients are frequently reporting Tinnitus as one of
the symptoms [6]. Therefore, it may be possible to
report significantly the potential onslaught or
prevalence of Tinnitus as a serious issue in SemD.
SemD evidence can support previous work
implicating of limbic network and a distributed
cortico-subcortical auditory in the pathogenesis of
these abnormal auditory percepts [6].
In general, Tinnitus is caused by peripheral and
central mechanisms such as peripheral injury, a
reorganization of central auditory pathways, or
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anomalies in the limbic system, which produce a
sensory emotional content experience [7]. There are
many different hypotheses to explain the reason of
Tinnitus. Some evidence suggests that Tinnitus can
be a pathophysiology that involves damage either
central or peripheral pathway or can be both [8].
Another Hypothesis proposed that Tonotopic maps
could be the cause of tinnitus. Tonotopic maps are
recognized in the auditory cortex and leads to a
sensation of Tinnitus frequencies [7]. Previously, it
was a suggested that the source of chronic Tinnitus
are coming by a compromised limbic corticostriatal
circuit that leads to a disordered in evaluation of
Tinnitus sensation’s perceptual and causing
disturbance in the control of cognition in the
tinnitus. [8]. On the other hand, anomalies related
to Tinnitus are inter-correlated between primary and
limbic auditory and/or two limbic area and indicates
necessity of auditory-limbic interactions in Tinnitus
as shown in figure 1. Although, the role of limbic
contributions nature to Tinnitus is not yet verified
[8]. Auditory-Limbic, shown in figure 1, clarifies
the interaction in Tinnitus, where the sensory input
is originated subcortically then enters both auditory
and limbic circuits via Medial Geniculate Nucleus
(MGN).
Medial Dorsal Nucleus (MDN),
Ventral Pallidum (VP),
amygdala (amyg),
Auditory Cortex (AC),
Medial Geniculate Nucleus (MGN).
Fig 1. Auditory-limbic interaction in Tinnitus [8].
Normally, sensory signal is defined by the limbic
system as perceptually irrelevant such as transient
Tinnitus followed by noise exposure. Then block
unwanted signal to MGN by projections from the
ventromedial Prefrontal Cortex (vmPFC) to the
auditory thalamic reticular nucleus (TRN, red
pathway). Therefore, an unwanted signal can be
reduced in either circuit. In the case of tinnitus,
inactive vmPFC output can prevent tinnitus signal
hosting and cortical thalamic activity. The structures
of thalamus in blue, and amygdala is noted in
lavender, basal ganglia noted in green, and cortical
are noted in gray. Mapping the hubs of the cortical
in Tinnitus it has been reported that having major
group differences across global networks, especially
in the gamma frequency band [4].
Typically, Tinnitus is usually associated with
damage to the hearing system such as chronic
exposure to noise or noise trauma [4]. This damage
can lead to drastic changes at different levels of the
central auditory system. As a result, it boosted the
rate of automatic fire and nervous synchronization
within the central auditory system. These changes
have been reported in both animal and human
studies and may be caused by various diseases [4].
Another suggestion about the cause of chronic
Tinnitus mentioned that it may be caused by plastic
reorganization in the auditory cortex followed by
peripheral deafferentation. Based on this hypothesis,
the reorganization process usually causes hair cells
to be lost in the inner ear; Sensorineural Hear
Losing (SNHL) may in some cases result in
cochlear injury due to sound trauma (i.e. exposure to
noise with a certain level of frequency band or
associated atrophy Age for the hair cell).
Moreover, the corresponding frequency range
can cause the lesion thresholds to rise. Furthermore,
the adjacent frequencies became more amplified due
to the expansion of the central representation in a
vacant frequency band [9]. Indeed, some of the
basic findings of Positron Emission Tomography
(PET) studies show that the frequencies
corresponding to the perceived Tinnitus frequencies
lead to frequency expansion in the auditory cortex
[9]. While tinnitus is usually considered a
heterogeneous condition, most patients who have
suffered from tinnitus have reported a complaint of
a sense of auditory weakness. With regard to the
brain mechanism of tinnitus sufferers, most current
data show a very important gray matter that shrinks
in the subcallosal region as shown in figure 2 [7].
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Fig 2. Gray-matter volume decreases in addition to
the changes throughout the whole brain [7].
Furthermore, it was found that there was an
expansion in the concentration of gray-matter in the
auditory thalamus of the Tinnitus group as shown in
figure 3 [7]. It is important to find that the structural
changes that associate with the region without
leaving the Tinnitus for different reasons like
activity in the artificial sub-region associated with
the unwanted auditory sensation that comes from
different amounts of disharmony symmetry,
especially in the area where the gray color is
shrinking the issue [7].
Fig 3. Gray-matter concentration increases [7].
Mainly, the role of the posterior thalamus and
subcallosal region in causing Tinnitus is combined
changes in both regions. In other words, these
changes seem to result in a tinnitus sensation.
Mulaw et al. (2005) A model suggested this:
1. Neural tinnitus activity is mainly in MGN and
results from reorganization after peripheral
hearing loss.
2. Inhibitory feedback from the subcallosal area
may help control nervous activity due to
Tinnitus.
3. Shrinkage of gray matter in the subcallosal
region may reduce these inhibitory reactions.
Because of this, people with peripheral hearing
loss may be at a health risk for tinnitus.
3 Results
A sound record was conducted in a workstation,
which located in an area where the operators could
perform paperwork or work on their computers with
tasks that were assigned to them. The recorded
signal time domain, frequency spectrum, and
spectrogram analysis are exhibited in figure 4-6
respectively. According to the time domain of the
sound (figure 4), sound pressure is bouncing around
0.05 Pa. However, acoustic (i.e., sound) pressure
level was plotted to show the noise pressure over
time. Refereeing to [1], the sound pressure level
(SPL) for the source of sound with pressure (p), is
defined as:
 

.. (1)
where pref= 20 10-6 Pa.
Therefore, the pressure of the sound at that
location could be exceeding the level of 60 dBA.
Therefore, it is not close to the maximum level of
the sound that is established by OSHA. Although
the noise level of the signal is not legally harmful,
due to the distance between the workstation and the
servers that causes a reduction in the SPL, it was
still representing a continuous distraction to the
workers in that server room.
A frequency spectrum analysis (figure 5) reveals
that the highest power of the signal is located in the
lower frequency band (i.e., 20–80 Hertz). At this
band, the power of the signal is ranging from -35 dB
to -60 dB. In figure 6, the signal power distribution
over the frequency is displayed in the time domain.
A frequency range of 0–120 Hz for the spectrogram
(figure 6) is evident in Figure 7. It shows that the
frequency band from 40–80 Hz has a level of power
that fluctuated between -50 dB to -60 dB. This range
is representing a gamma frequency range, which is
considered as the most harmful range in terms of
hearing impairment.
As a result, employees were exposed to a high-
power level of continuous gamma frequency sound
while spending the majority of their work shift in
the server room to perform daily tasks and monitor
the network. As mentioned earlier, long-term
exposure in this frequency range may cause
significant damage to the hearing system such as
chronic noise trauma [4].
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Fig 4. Time domain for the sound pressure.
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Fig 5. Frequency spectrum for the noise signal.
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Fig 6. Frequency spectrum in time domain for the noise signal.
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Fig 7. Spectrogram for noise signal.
4 Conclusion
In this paper, it was hypothesized that high sound
level at low frequency can cause a deleterious effect
on individuals who exposed to it for extended
periods. Some evidence supports this theory, and
this paper summarizes similar facts. The purpose
was to promote more investigative work in this area
and facilitate this discussion.
The impact of noise on job performance should
not rely on sound level only as parameter. It should
look up the frequency range of the noise too, as the
frequency can influence a person’s hearing ability
and even mental functionality. According to this
research effort, it is recommended that employees in
work environments similar to the server room or
data center to wear personal protective equipment
until an appropriate engineering solution can be
implemented to reduce noise levels.
Finally, it is recommended to collect more data
from inside server rooms with different
configurations, which help in investigation and
validation the impact of this sort of noise on human
performance in occupational environments.
References:
[1] Sultan, H., Katiyar, A., & Smruti , S. R., Noise
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[2] Bryant, R. E., & O’Hallaron, D. R., Computer
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[3] OSHA. Occupational noise sxposure.
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[6] Mahoney, C. J., Rohrer, J. D., Goll, J. C., Fox,
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Contribution of individual authors to
the creation of a scientific article
(ghostwriting policy)
Ahmed N. H. Alnauimy carried out the data
collection and statistical analysis.
Michael Johnson assisted in data collection.
Rasha Hashem assisted in the Statistics.
Creative Commons Attribution License 4.0
(Attribution 4.0 International, CC BY 4.0)
This article is published under the terms of the Creative
Commons Attribution License 4.0
https://creativecommons.org/licenses/by/4.0/deed.en_US
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